Patrick carroll breast cancer



Indeed, MondoA knockdown impaired the ability of N-Myc-overexpressing Tet21N cells to form colonies in soft agar and to form tumors in mouse xenograft models. Follow Us Fundraise. The expression of these genes was also dependent on the MondoA dimerization partner Mlx, indicating a requirement for MondoA-Mlx heterodimers in upregulating metabolism in the context of N-Myc overexpression.


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Myc requires MondoA to reprogram cancer metabolism

Overexpression of Myc-family TFs leads to alterations in gene transcription that influence numerous cellular processes, including metabolism, to facilitate increased cellular growth. The expression of these genes was also dependent on the MondoA dimerization partner Mlx, indicating a requirement for MondoA-Mlx heterodimers in upregulating metabolism patrick carroll breast cancer the context of N-Myc overexpression. These findings also open new possibilities for therapy of Myc-overexpressing cancers.

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High expression of the signature was significantly correlated with poor prognosis in all cases to differing degrees. MondoA depletion also reduced the viability of several human cancer cell lines overexpressing c-Myc, indicating a general requirement for MondoA in c-Myc-induced cell growth. We encourage individuals with diverse backgrounds to apply and desire priority referrals of protected veterans. Overexpression of Myc-family TFs leads to alterations in gene transcription that patrick carroll breast cancer numerous cellular processes, including metabolism, to facilitate increased cellular growth. Indeed, MondoA knockdown impaired the ability of N-Myc-overexpressing Tet21N cells to form colonies in soft patrick carroll breast cancer and to form tumors in mouse xenograft models.

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